QUOTE(Masiukiene @ 2008 09 12, 11:51)
Jo dėl tiroksino tai man sumažino, nes po paskutinių tyrimų sakė ,kad labai suspaustas (ką reiškia nežinau) bet matyt vistiek per daug nes mane dar smaugia atrodo kad neoperuota, gal dėl to ir svoris krenta, taip džiaugiaus ,kad 4kg priaugau ,o dabar vėl toj pačioj vietoj.Dirbu tai daug gal irgi turi įtakos , nes kai pavargsti tai jau nieko nebesinori net valgyt
man irgi sumazino doze iki 150, nors 'pagrazejau' virs 10 kg...
'Suspaustas [suppression]', mano galva, kalbama apie per maza TTH, nors ji po Ca operacijos pirmuosius 3-5 metus rekomenduojama palaikyti maza. Va cia info - pasiskaitykite, as pabandziau palikti tik esme, taigi atsiprasau, jeigu kur nors rysio truks.
* The role of TSH-suppressive therapy after initial
treatment is twofold: the first aim is to correct the hypothyroidism using a dosage appropriate to
achieve normal blood levels of thyroid hormone.
The second aim is to inhibit the TSH-dependent
growth of residual cancer cells by decreasing the
serum TSH level to %0.1 mU/l (63, 64). In patients
considered in complete remission at any time during
follow-up, there is no need to suppress endogenous
TSH and thus therapy may be shifted from
suppressive to replacement (65).
monitored every 612 months.
* TSH-suppressive therapy (serum TSH %0.1 mU/l) is
mandatory in patients with evidence of persistent
disease (including detectable serum Tg and no other
evidence of disease). In high-risk patients who have
achieved apparent remission after treatment, suppressive
therapy is advised for 35 years. In low-risk
patients, when a cure has been assessed, the risk of
subsequent recurrence is low (!1%) and the dose of
LT4 can be immediately decreased, aiming for a serum
TSH level within the lower part of the normal range
(between 0.5 and 1.0 mU/l) (66).
Lina, cia tau, juk tu skundiesi sirdimi: However, in elderly
patients and in
patients with known cardiac disease,
TSH suppression should be avoided. During subclinical
thyrotoxicosis an additional matter of concern is the
evidence that the majority of patients have a
prothrombotic profile (68).
* In the case of documented stable remission, the
optimal TSH level should be in the low-normal range,
but, if the woman has persistent disease or is at high
risk of recurrence, serum TSH should be kept
suppressed at around 0.1 mU/l.